黄瓜视频软件不经过脱硫时，当打开开关时，硫化氢气体对人呼吸道有刺激性，长期受此刺激，可能引起呼吸道粘膜炎。硫化氢对黄瓜视频软件炉具及其配件和家用电器（含金属）有腐蚀作用，容易损坏。

　　When biogas is not desulfurized, hydrogen sulfide gas can be irritating to the human respiratory tract when the switch is turned on. Prolonged exposure to this stimulation may cause respiratory mucositis. Hydrogen sulfide has a corrosive effect on biogas stoves and their accessories, as well as household appliances (including metals), and is prone to damage.

　　发生H2S中毒的特点：H2S最大的危害是意外接触造成突然死亡。不能根据臭鸡蛋味来判断作业场所的是否存在H2S和H浓度。

　　Characteristics of H2S poisoning: The greatest harm of H2S is sudden death caused by accidental contact. It is not possible to determine the presence of H2S and H concentration in the workplace based on the smell of rotten eggs.

　　发病机制：血中高浓度H2S可直接刺激颈动脉窦和主动脉区的化学感受器，导致反射性呼吸抑制。H2S可直接作用于脑，低浓度起兴奋作用，高浓度起抑制作用，引起昏迷、呼吸中枢和血管运动中枢麻痹。因HS是细胞色素氧化酶的强抑制剂能与线粒体内膜呼吸链中的氧化型细胞色素氧化酶中的三价铁离子结合，而抑制电子传递和氧的利用，引起细胞内缺氧，造成细胞内窒息。因脑组织对缺氧最敏感，故最易受损以上两种作用发生快，均可引起呼吸骤停，造成突然死亡。在发病初如能及时停止接触，则可因H在体内很快氧化失活而迅速和完全恢复。

　　Pathogenesis: High concentrations of H2S in the blood can directly stimulate chemoreceptors in the carotid sinus and aortic region, leading to reflexive respiratory depression. H2S can directly act on the brain, with low concentrations having excitatory effects and high concentrations having inhibitory effects, causing coma, paralysis of the respiratory and vascular motor centers. HS is a strong inhibitor of cytochrome oxidase that can bind to trivalent iron ions in the oxidative cytochrome oxidase in the mitochondrial inner membrane respiratory chain, inhibiting electron transfer and oxygen utilization, causing intracellular hypoxia and suffocation. Due to its sensitivity to hypoxia, brain tissue is most susceptible to damage. Both of these effects occur quickly and can cause respiratory arrest, leading to sudden death. If contact can be stopped in a timely manner at the beginning of the disease, it can be quickly and completely restored due to the rapid oxidation and inactivation of H in the body.

　　硫化氢的特性及不同浓度下的症状?

　　What are the characteristics of hydrogen sulfide and the symptoms at different concentrations?

　　浓度为1ppm~10ppm时，允许暴露8小时；浓度为20pm时，必须戴适当的防护器具；浓度为50~70ppm时，刺激呼吸道；浓度为100ppm时，2-15分钟內丧失嗅觉，可能引起喉咙灼痛、头痛和恶心浓度为200pm时，嗅觉很快丧失，眼、喉咙灼痛；浓度为1000m以上时，立即引起昏迷，除非立即抢救，否则就会死亡。

　　When the concentration is 1ppm~10ppm, exposure for 8 hours is allowed; When the concentration is 20pm, appropriate protective equipment must be worn; When the concentration is 50-70ppm, it irritates the respiratory tract; When the concentration is 100ppm, the sense of smell is lost within 2-15 minutes, which may cause burning sensation in the throat, headache, and nausea. When the concentration is 200pm, the sense of smell is quickly lost, and there is burning sensation in the eyes and throat; When the concentration is above 1000m, it immediately causes coma, and unless rescued immediately, death will occur.

　　H2S中毒的早期护理注意事项：

　　Early nursing precautions for H2S poisoning:

　　1.在中毒者心跳停止之前，转移到空气新鳟的地方直至恢复正常呼吸。

　　1. Before the poisoned person's heartbeat stops, move to a place with fresh air until normal breathing is restored.

　　2.如果眼睛受到伤害，可用清水清洗，然后作冷敷护理。

　　If the eyes are injured, they can be washed with clean water and then treated with cold compress.

　　3.血中高浓度H2S可直接刺激颈动脉窦和主动脉区的化学感受器，导致反射性呼吸抑制。

　　3. High concentrations of H2S in the blood can directly stimulate chemoreceptors in the carotid sinus and aortic region, leading to reflexive respiratory depression.

　　4.HS可直接作用于脑，低浓度起兴奋作用，高浓度起抑制作用，引起昏迷、呼吸中枢和血管运动中枢麻痹。因Hs是细胞色素氧化酶的强抑制剂，能与线粒体内膜呼吸链中的氧化型细胞色素氧化酶中的三价铁离子结合，而抑制电子传递和氧的利用，引起细胞内缺氧，造成细胞內室。因脑组织对缺氧最敏感，故最易受损。

　　4. HS can directly act on the brain, with low concentrations having excitatory effects and high concentrations having inhibitory effects, causing coma, paralysis of the respiratory and vascular motor centers. As Hs is a strong inhibitor of cytochrome oxidase, it can bind with trivalent iron ions in the oxidative cytochrome oxidase in the mitochondrial inner membrane respiratory chain, inhibiting electron transfer and oxygen utilization, causing intracellular hypoxia and creating an intracellular compartment. Because brain tissue is most sensitive to hypoxia, it is most susceptible to damage.

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